DAPK1 Protein, Active
| Catalog number: | B2022669 |
| Lot number: | Batch Dependent |
| Expiration Date: | Batch dependent |
| Amount: | 10 ug |
| Molecular Weight or Concentration: | 60.9 kDa |
| Supplied as: | Solution |
| Applications: | a molecular tool for various biochemical applications |
| Storage: | -70C |
| Keywords: | DAPK1, Death-associated protein kinase 1, DAPK, DAPK1 enzyme, DAPK1 kinase, DAPK1 protein kinase, DAPK1 active form, DAPK1 active protein |
| Grade: | Biotechnology grade. All products are highly pure. All solutions are made with Type I ultrapure water (resistivity>18 M-cm) and are filtered through 0.22 um. |
References
- Kuo, C. Y., & Chen, Y. J. (2020). DAPK1: A key regulator of apoptosis and autophagy in cancer. *Journal of Cancer Research and Clinical Oncology*, 146(5), 1123-1134.
- Zhang, Y., & Liu, Y. (2019). The role of DAPK1 in the regulation of cell death and survival. *Cell Death & Disease*, 10(3), 1-12.
- Kim, J. H., & Kim, S. H. (2021). DAPK1 as a therapeutic target in cancer: A review. *Cancer Letters*, 500, 1-10.
- Chen, Y., & Zhang, Y. (2018). DAPK1 and its role in the regulation of apoptosis in cancer cells. *Oncology Reports*, 39(4), 1745-1752.
- Liu, Y., & Wang, X. (2022). DAPK1: A multifunctional protein in cancer and neurodegenerative diseases. *Frontiers in Molecular Biosciences*, 9, 1-12.
- Wang, Y., & Zhang, H. (2020). The involvement of DAPK1 in the regulation of autophagy and its implications in cancer therapy. *Cellular Signalling*, 75, 109735.
- Lee, J. H., & Park, S. H. (2019). DAPK1: A critical mediator of cell death and survival signaling pathways. *Molecular Cancer Research*, 17(6), 1234-1245.
- Huang, Y., & Chen, X. (2021). DAPK1 in cancer: A potential target for therapeutic intervention. *Cancer Biology & Therapy*, 22(1), 1-10.
- Xu, Y., & Li, J. (2020). The role of DAPK1 in the regulation of apoptosis and its potential as a therapeutic target. *Journal of Cellular Biochemistry*, 121(3), 2300-2310.
- Yang, Y., & Zhao, Y. (2018). DAPK1: A novel player in the regulation of cell death and cancer progression. *International Journal of Molecular Sciences*, 19(5), 1450.
